urate

1. Effects of Gouty Granule on LP-PLA_2 Value and Swollen Ankle Joint Degrees of Rat Model of Sodium Urate Arthritis
   痛风颗粒对尿酸钠关节炎模型大鼠踝关节肿胀度、血清LP-PLA2的影响
2. Effect of uric acid on the expression of human urate transporter gene in HK-2 cells
   尿酸对肾小管上皮细胞人尿酸盐转运子基因表达的影响
3. Effect of Tong Feng Ning ( TFN) on Monoamine Neurotransmitters in Hypothalamus in Rats of Monosodium Urate ( MSU) Induced Arthritis
   痛风宁对尿酸钠致大鼠关节炎下丘脑单胺类神经递质的影响
4. Advances in the Research of Xanthine Oxidase Inhibitors for Lowering Urate
   降尿酸药物黄嘌呤氧化酶抑制剂的研究进展
5. The findings raise the possibility that urate might help protect brain cells from damage by toxic molecules known as free radicals, says Brian Fiske of the Michael J.
   尿酸可能降低活性自由基的水平使之没有机会伤害重要的脑细胞。
6. To Discuss the Mechanism of Vascular Endothelial Cell Damage by Urate
   尿酸盐致血管内皮细胞损伤的机制探讨
7. High levels of a natural substance in the blood called urate might slow the progression of Parkinson's, a study out today suggests.
   研究表明血中体内天然产物尿酸过高可能会减缓帕金森病的发展进程。
8. The chemical urate, which is known to cause gout, appears to slow the progression of Parkinson's disease, US researchers have concluded.
   美国研究人员推断，可以引起痛风的化学物尿盐酸，似乎对帕金森病的发展有缓和作用。
9. Result: Saluresis included phosphate, oxalate and urate, et al.
   结果：尿盐分别为磷酸盐、草酸盐、尿酸盐等。
10. The diagnosis was based on the severe hyperuricemia, oliguria, acute deterioration of renal function, and urate crystal deposition in the renal collecting ducts in renal biopsy.
    肾脏切片显示出尿酸结晶堆积于收集管中、诊断为急性尿酸性肾病变。
11. And the true masterpiece, the mahogany urate lornis.
    而真正杰出的是红褐色的尤瑞特罗尼斯。
12. Study on the relationship among the level of plasma urate and onset and prognosis in cerebral infarction caused by different etiological subtype
    血尿酸水平与不同病因分型脑梗死发病及预后的关系
13. Tophaceous gout results from continued precipitation of sodium urate crystals during attacks of acute gout.
    沙粒状痛风是由急性痛风发作期尿酸钠盐结晶继续析出引起。
14. Curative Effect of Xi's Gout Killer-2 on the Experimental Rats of Urate Nephropathy
    奚氏痛风灵2号对慢性高尿酸血症肾病大鼠尿酸代谢的影响
15. The main characters of pathological changes were bursal dropsy like jelly and urate aggradation in kidney, probabilities of which were 67.2% and 73.11% respectively. The other pathological changes were not typical.
    病变以法氏囊呈胶胨样水肿和肾脏尿酸盐沉积为主要特征，二者出现率分别占67.2%和73.11%，其它病变多不典型；
16. Trinder method was used to detect the serum urate levels.
    采用Trinder法测定大鼠血清尿酸值。
17. Low-dose Chinese herbs group can effectively decline the serum urate levels.
    中药低剂量组可以降低血尿酸。
18. The detection of urate crystal by synovium and synovial fluid is the key basis for diagnose.
    根据临床表现、生化检查及X射线检查即可确诊，滑膜或关节液检查到尿酸盐结晶是诊断的重要依据。
19. Most prokaryote and eukaryote have intact urate oxidase gene ( wo) that produces urate oxidase activity and excretes allantoin as the end product of purine metabolism.
    大多数的原核和真核生物都有完整的尿酸氧化酶基因，产生具生物活性的尿酸氧化酶，以尿囊酸作为代谢最终产物。
20. Experimental Study on Clinical Pathology of Urate Deposition in Chickens
    鸡尿酸盐沉积临床病理学实验研究
21. The animal model and sodium urate solution were self prepared.
    制备尿酸钠溶液及动物造模。
22. Age, profession, smoking and urine pH have no correlation with urate nephropathy.
    年龄、职业、吸烟及住院期间尿pH值与痛风性肾病无显著相关。
23. Crystal was collected from gout tophi. It had confirmed that are monosodium urate crystals by polarizing microscope.
    方法1.采集痛风病人痛风结节中的尿酸盐晶体，应用偏振光显微镜观察晶体形态，以证实其结晶为尿酸盐结晶。
24. Uric acid nephropathy ( UAN) also known as gouty nephropathy is due to excessive concentrations of uric acid urate deposition in the kidney, kidney damage caused by the crystallization.
    尿酸性肾病（UricacidNephropathy，UAN）又称痛风性肾病，是由于血尿酸浓度过高，尿酸盐在肾脏沉积、结晶导致的肾脏损伤。
25. Urate oxidase has been found in the body of many species, while birds and some higher primates ( eg apes and humans) lack functional urate oxidase in vivo and excrete uric acid as the end product of purine metabolism.
    在许多物种体内均发现有尿酸氧化酶，但在鸟类及一些高等灵长类动物（如猿和人类）的体内却缺乏具有功能的尿酸酶，而是以尿酸作为嘌呤代谢的终产物排出体外。
26. Human beings lack active uricase because of gene mutations, leading to uric acid as the end product of purine metabolism and resulting in the development of urate nephropathy and gouty arthritis when the concentration of uric acid reaches its solubility limit in blood ( hyperuricemia).
    人类由于基因突变而缺乏活性尿酸酶，这导致人类以尿酸作为嘌呤代谢的终产物。当人血液中尿酸浓度高于其溶解度（高尿酸血症）时可引起尿酸性肾病和痛风性关节炎。
27. Studies have shown that urate oxidase can far superior in reducing uric acid levels than other drugs in scale and speed.
    有研究表明，尿酸氧化酶降低体内尿酸水平的幅度和速度远远优于其它药物。
28. In this study, we take advantage of several silkworm body color mutant strains to investigate deposition patterns and biological mechanism of urate and melanin in the integuments of these mutant larvae.
    本研究利用几种家蚕体色突变体来研究黑色素和尿酸在表皮中的沉积模式并探索沉积模式的生物机制。
29. It has been reported that the urate oxidase with PEG-modified can reduce its immunogenicity, but enzyme activity has decreased greatly after PEG modified.
    已有研究通过PEG化修饰尿酸氧化酶以降低其免疫原性，但经PEG修饰后酶的催化活性下降幅度较大。
30. The urate crystal appeared in urine is a marker of uric acid in super-saturation. It is very important to detect urate crystal in urine of Gout patient for diagnosis and assessment of Gout. 3.
    尿尿酸盐晶体是尿酸在尿液中处于过饱和状态的标志，通过检测痛风患者尿液尿酸盐结晶，探讨痛风患者尿液尿酸盐结晶对痛风早期诊断和评价病情的临床意义。

noun

1. a salt of uric acid